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. ·The interplay between the mechanisms involved in the activation of the alpha2c-adrenergic receptor by CGP12177 and isoproterenol in the human placenta.
CGP12177 is a long-acting agonist for the alpha2c-adrenergic receptor that has been proposed as a potential treatment for preterm labor. However, the most appropriate dosage and route of administration of CGP12177 to prevent preterm labor needs to be determined. Therefore, we compared the effects of CGP12177 and isoproterenol on the contraction of human placental tissues in vitro and investigated whether stimulation of the alpha2c-adrenergic receptor by CGP12177 is inhibited by preincubation with the beta1- and beta2-adrenergic receptor blocking agents, ICI 118,551 and ICI 89,406. Placentas from women at term undergoing elective cesarean sections (n=6) were used for this study. The presence of beta-adrenergic receptors and alpha2c-adrenergic receptors was demonstrated in trophoblast cells and syncytiotrophoblast by the specific binding of (125)I-iodocyanopindolol (ICYP) and (125)I-iodocyanopindolone (ICYNP), respectively. The anticholinergic agent atropine sulfate was used to assess the cholinergic mediation of the contraction. Both CGP12177 and isoproterenol stimulated a concentration-dependent contraction of human placental tissues. CGP12177 was equipotent to isoproterenol in eliciting a maximum contraction at 100 microM. Preincubation of tissues with the beta-adrenergic receptor blocker ICI 89,406, but not with ICI 118,551, abrogated the contractile response to CGP12177. Preincubation of the tissues with ICI 89,406 but not with ICI 118,551 partially prevented the contraction to isoproterenol but had no effect on the contraction induced by atropine. The results support the hypothesis that CGP12177 induces the contraction of human placental tissues by acting through the alpha2

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